Schilling Long-Hauler

Manual for Long-Covid and Post-Vaccine Syndrome
1. Auflage 2022
ISBN: 978-3-347-68013-5
Verlag: tredition
Format: EPUB
Kopierschutz: 6 - ePub Watermark

E-Book, Englisch, 436 Seiten

Manual for Long-Covid and Post-Vac: How do they work, which are the problems? How can they be detected and diagnosed? Which investigations and lab markers are crucial? What can we learn from them? Which tools do we have, to get back our health - may they be drugs, supplements, micronutrients, nutrition or instrument-based? The book provides comprehensive but comprehensible explanations, protocols and procedures.

Autoren/Hrsg.

Schilling, Florian

Leseproben

Infection vs. Vaccination: Immunological effect

To understand what happens with LC or PVS, it is essential to understand the mechanisms and processes underlying a Covid-19 infection or vaccination.

Vaccination immunologically resembles a severe course

The ingestion of a pathogen is referred to as contagion.. Only if the pathogen manages to multiply in the body does a contagion turn into an infection. . The PCR test does not distinguish between the two stages, so PCR+ does not automatically mean being infected. In the case of SARS-CoV2 (SC2), the cells of the upper respiratory tract are infected first (mouth, nose, and throat). In particular with omicron, this is already the end in most cases; no further spread takes place. If it does, the virus next reaches the bronchial tubes (bronchitis, equivalent to an influenza infection), and in the rare case of a severe course, also the lungs (Pneumonia). The latter affected about 10% of Covid 19 patients in Delta, while Omicron is still at 1%.

Complicated courses are now characterized by the virus breaking through the primary barrier of the respiratory tract and entering the bloodstream. From there, it can spread throughout the body (viremia) resulting in life-threatening complications: Infestation of internal organs, excessive inflammation (Hyperinflammation, „cytokine storm„), uncontrolled clotting (DIC, „ disseminated intravascular coagulation „) und and, in the worst case, multi-organ failure. .

The appearance of intact, replicable and spike-bearing SC2 viruses in the bloodstream, especially in large numbers, is the exceptional case in the context of a natural infection.

The situation is different now with vaccination: the vaccine is injected intramuscularly and thus enters the body at a completely different site than the virus does during an infection. Important: the vaccine does not remain at the injection site (this is a modern fairy tale that is still spread by numerous public actors), but spreads throughout the organism within hours. This fact has already been established by the manufacturers during the registration studies and can be read in official documents of the registration authorities. For example, the vaccine RNA was detectable in the lungs, liver, spleen, ovaries, and testicles, but also in the brain.1,2

Since all cells can be reached by the vaccine, all cells that take up the vaccine (a) incorporate it into their cell wall and (b) release it to the outside, there is a massive and systemic appearance of the spike protein throughout the body during vaccination. Thus, in this respect, vaccination corresponds to a severe course with viremia. .

In fact, it has now been shown that the concentration of spike protein in the bloodstream of vaccinated individuals is above that which can be measured during an acute infection.3 Because of the enormous toxicity of spike protein, acute complication risks after vaccination are comparable to those of critically ill patients in hospital. Not surprisingly, clotting disorders and myocarditis (inflammation of the heart muscle) occur in many cases - and this is only the tip of the iceberg. A vaccination that would simulate a simple contagion (which would be quite sufficient to build immunity) would have to look like this:

• Spraying of the antigen (e.g., of the killed pathogen = dead vaccine) into the nasopharynx

• Processing of this antigen by the local immune system

• Formation of cellular and humoral (antibody-based) immunity

Advantage: The toxic spike would not appear in the bloodstream, the processing of the information by the immune system would correspond 100% to the natural course. Instead, the immune system is confronted by the current vaccines from 0 to 100 with a, from its point of view, potentially lethal situation. Accordingly, the immune system reacts massively … and the collateral damage can be considerable. Which brings us directly to the next point - what kind of immunity is created by infection or vaccination in the first place.

Vaccination and infection produce a different kind of immunity.

Basically, our immune system can be divided very simply with two criteria. First, there is the distinction between cellular and humoral, the last being based on defence proteins. In addition, there is the innate and the adaptive (adaptive, i.e., capable of learning) defence. We have the former from birth, it is always available - but it cannot specialize to specific opponents, so it does not form an immunological memory. The adaptive defence is much more potent and effective - but must first study an opponent before it can fight him. Therefore, it is useless during the first contact with a previously unknown enemy. Only after some time (about 1-2 weeks) it has adapted and can then effectively intervene in the action. Thus, we have innated and adaptive defence cells as well as innated and adaptive defence proteins:

Innated

adaptive

cellular

Granulocytes monocytes / macrophages natural killer cells dendritic killer cells mast cells

T-Lymphocytes • Killer cells • Helper cells • Memory cells • Suppressor cells B-Lymphocytes • Plasma cells • Memory cells

humoral

Complementary system

Antibodies

FIGURE 1: CLASSIFICATION OF THE IMMUNE SYSTEM.

When in the course of a contagion or infection, the adaptive defence specializes, this leads to a repertoire of possible tools to fight the enemy in the future. While on the side of the cellular defence, specific T-killer cells emerge - which can then recognize and destroy infected cells (melee division) - the antibody response fans out to different antibody classes, each with different tasks and strengths:

• First line of defence: In respiratory pathogens, IgA- type antibodies are the most important. They are deposited on the mucous membranes (respiratory and digestive tract) and represent a kind of minefield for the pathogen in the future: As soon as the pathogen settles on the spot, it is neutralized by the antibodies - even before it can penetrate further into the cells and the organism. IgA thus prevent infections and contribute to sterile immunity.

• IgM are only formed as an immediate reaction, circulate in the blood, and are replaced by IgG within 2 weeks:

• Last line of defence: IgG antibodies circulate in the bloodstream and thus represent the third line of defence. Thus, should the pathogen make it into the bloodstream, IgG- antibodies prevent the worst - viremia or a septic course.

FIGURE 2: THE THREE BARRIERS OF THE ADAPTIVE DEFENSE AGAINST RESPIRATORY PATHOGENS; FROM LEFT TO RIGHT: IGA ON THE MUCOSA, T-KILLER CELLS MONITORING THE MUCOSAL CELLS, IGG IN THE BLOODSTREAM.

For complete immunity, especially sterile immunity (the pathogen cannot be passed on to third parties in case of contagion), we need all three levels - IgA, T-killer cells, and IgG. In the course of a natural infection, there is also a possibility (and a high probability) that all three components are formed. In the case of vaccination, on the other hand, a strongly IgG-heavy immunity develops. In the case of renewed contact with the pathogen in the respiratory organs, however, this is of little use - IgG antibodies simply do not reach the respiratory organs. If we imagine the interaction of these defence components like a military battle, the distribution of tasks would be something like this:

• IgA antibodies form an upstream minefield that the opponent must first overcome.

• This is followed by a defence line of excellently trained close combatants - the T killer cells.

• In the background, reserves are kept ready to hold off opponents who have broken through and to prevent total defeat: IgG antibodies.

Known consequence from what has been described: Corona vaccinated individuals are not protected against infection and can still pass on the pathogen. Herd immunity is therefore not achievable with current vaccines. Extraneous protection as an argument for vaccination does not exist. The clear inferiority of vaccine immunity to natural immunity has been demonstrated many times. Naturally acquired immunity is up to twenty-seven times more effective than "vaccinated" immunity.4 It also lasts significantly longer; while vaccination no longer provides protection after a few months, natural immunity lasts for decades, not infrequently for a lifetime. Even in the 21st century, immunity has been demonstrated in Spanish flu survivors - over 80 years later!

What is the reason for this? Well, the vaccinations present as an enemy not the actual virus (this would significantly trigger the killer cells), but a viral protein - the spike. The humoral defence, i.e., the antibody defence, is specialized in the defence against hostile, foreign proteins. Moreover, since the vaccine spike appears in the organism, in the bloodstream, and not on the interfaces with the environment (i.e., the mucosa), the formation of IgG is stimulated - not the formation of IgA. So, we already have...